Scientists at the Clinical Hospital of Barcelona in collaboration with the University of Santiago de Compostela and the Catholic University of Leuven (Belgium) announced on Tuesday that, according to a joint study, the brain has the ability to control the composition of intestinal bacteria and thus influence the feeling of satiety.
“The brain communicates with the intestine to indicate whether or not, it has the ability to control the composition of bacteria in the intestine in record time, 2 hours, and therefore influence the sensation, or not, of satiety,” said a press release issued on Tuesday by the Belgian University.
The interest of the study lies in demonstrating that, by restoring communication between the brain and the intestine, “we could act on intestinal bacteria and control our eating habits.”
“Normally, the areas that control the appetite in the brain (hypothalamus) light when we are hungry and go out when the body is full, like an ignition and off switch. When this area is turned off, the body consumes its own energy reserves, which helps regulate the weight,” the press release clarified.
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Scientists show that the brain can control appetite
However, the study revealed that in people with type 2 diabetes this system “works badly” and satiety information “is not transmitted correctly, which explains a tendency to obesity.”
In their research, scientists used genetic and pharmacological techniques to study brain areas that control appetite.
As a result, they observed that when the area that inhibits food intake is activated or blocks a “ultra -grape” change, in two hours, in the composition of the intestinal microbiota.
“In summary, modifying the areas that control appetite or hunger in the brain has an impact on the bacteria of the intestine, which react as if they had received nutrients, although no food has been ingested. As a result, they send messages to the brain telling him that the body is full or has not received any food, when in reality this is not so,” he said.
This discovery “will ultimately allow intervention processes to restore communication between the brain and the intestine and thus influence eating habits.”
With EFE information.
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